Luciana Venturini Rossoni

Principal Investigator (PI)

Associate Professor

Positions

Brief Bio

Medical doctor at the “Santa Casa de Misericórdia” School of Medicine in Vitória (1996) and PhD in Physiological Sciences, in Cardiovascular Physiology, at Federal University of Espirito Santo (2001), with an internship period abroad at the Pharmacology Department of the Faculty of Medicine at Universidad Autónoma de Madrid, Spain. Postdoctoral (2008-2009) at The University of Manchester, UK, working as an associate researcher with the Cardiovascular Research Group. Since 2002, professor at the Department of Physiology and Biophysics, Biomedical Science Institute at University of Sao Paulo.

Since my undergraduate training at the medical school, I started to research the cardiovascular system at the Dr Vassallo Lab (UFES-Brazil). My interest in vascular function and dysfunction in cardiovascular diseases took me to develop my graduate training at the laboratory of Dr Vassallo and Dr Jesus Marín and Mercedes Salaices (UAM-Spain) studying the role of ouabain on the vascular reactivity adjustments observed in hypertension. During my postdoctoral training I worked in Dr Heagerty group (Cardiovascular Research Group, UK) using human and animal models to evaluate vascular function by pressure myograph system. I have been PI of my own laboratory (LabVasc) since 2002, at the Physiology and Biophysics Department, Biomedical Science Institute at University of Paulo – Brazil. During all this time, graduate and undergraduate students have been participating to the Lab. My group has won fellowships, travel, and presentation awards, and we consistently received financial support to develop our research (FAPESP, CNPq and CAPES). The main aim of the Vascular Physiology Lab is to understand cell signaling pathways and the molecular bases involved in functional (vascular reactivity) and structural (structural and mechanical remodeling) adjustments of conductance and resistance vessels in cardiovascular diseases (hypertension and heart failure), as well as in pharmacological and non-pharmacological maneuvers (exercise training, microbiota and cross-fostering) that can restore or prevent vascular damage.